ZNF326 Antibody (N-term)
Affinity Purified Rabbit Polyclonal Antibody (Pab)
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
Application ![]()
| WB, E |
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Primary Accession | Q5BKZ1 |
Other Accession | O88291, F1MJM0, NP_892021.1 |
Reactivity | Human |
Predicted | Bovine, Mouse |
Host | Rabbit |
Clonality | Polyclonal |
Isotype | Rabbit IgG |
Calculated MW | 65654 Da |
Antigen Region | 127-155 aa |
Gene ID | 284695 |
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Other Names | DBIRD complex subunit ZNF326, Zinc finger protein 326, Zinc finger protein interacting with mRNPs and DBC1, ZNF326, ZIRD |
Target/Specificity | This ZNF326 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 127-155 amino acids from the N-terminal region of human ZNF326. |
Dilution | WB~~1:1000 |
Format | Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification. |
Storage | Maintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles. |
Precautions | ZNF326 Antibody (N-term) is for research use only and not for use in diagnostic or therapeutic procedures. |
Name | ZNF326 |
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Synonyms | ZIRD |
Function | Core component of the DBIRD complex, a multiprotein complex that acts at the interface between core mRNP particles and RNA polymerase II (RNAPII) and integrates transcript elongation with the regulation of alternative splicing: the DBIRD complex affects local transcript elongation rates and alternative splicing of a large set of exons embedded in (A + T)-rich DNA regions. May play a role in neuronal differentiation and is able to bind DNA and activate expression in vitro. |
Cellular Location | Nucleus matrix. |

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Provided below are standard protocols that you may find useful for product applications.
Background
Probable transcriptional activator which may play a role in neuronal differentiation. Able to bind DNA and activate expression in vitro (By similarity).
References
O'Donnell, C.J., et al. BMC Med. Genet. 8 SUPPL 1, S4 (2007) :
Andersen, J.S., et al. Nature 433(7021):77-83(2005)

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