LMO4 Antibody (Center)
Affinity Purified Rabbit Polyclonal Antibody (Pab)
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
Application
| WB, IF, FC, E |
---|---|
Primary Accession | P61968 |
Other Accession | P61969, Q3SWZ8, NP_006760.1 |
Reactivity | Human, Mouse |
Predicted | Bovine |
Host | Rabbit |
Clonality | Polyclonal |
Isotype | Rabbit IgG |
Calculated MW | 17994 Da |
Antigen Region | 104-131 aa |
Gene ID | 8543 |
---|---|
Other Names | LIM domain transcription factor LMO4, Breast tumor autoantigen, LIM domain only protein 4, LMO-4, LMO4 |
Target/Specificity | This LMO4 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 104-131 amino acids from the Central region of human LMO4. |
Dilution | IF~~1:10~50 WB~~1:1000 FC~~1:10~50 |
Format | Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification. |
Storage | Maintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles. |
Precautions | LMO4 Antibody (Center) is for research use only and not for use in diagnostic or therapeutic procedures. |
Name | LMO4 |
---|---|
Function | Transcription cofactor. Plays a role in establishing motor neuron identity, in concert with MNX1, acting, at least in part, to disrupt LDB1-LHX3 complexes thereby negatively modulating interneuron genes in motor neurons. |
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Provided below are standard protocols that you may find useful for product applications.
Background
This gene encodes a cysteine-rich protein that contains two LIM domains but lacks a DNA-binding homeodomain. The encoded protein may play a role as a transcriptional regulator or as an oncogene.
References
Tian, Y., et al. Clin. Exp. Metastasis 27(7):455-463(2010)
Ribases, M., et al. Biol. Psychiatry 66(10):926-934(2009)
Montanez-Wiscovich, M.E., et al. Oncogene 28(41):3608-3618(2009)
Huang, Z., et al. Dev. Biol. 327(1):132-142(2009)
Caporaso, N., et al. PLoS ONE 4 (2), E4653 (2009) :
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