Anti-FTO (Fat mass and obesity related protein) Antibody
Our Anti-FTO (Fat mass and obesity related protein) primary antibody from PhosphoSolutions is mouse
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
Application ![]()
| WB |
---|---|
Primary Accession | Q9C0B1 |
Reactivity | Bovine |
Host | Mouse |
Clonality | Monoclonal |
Isotype | IgG2a |
Clone Names | 5-2H10 |
Calculated MW | 58282 Da |
Gene ID | 79068 |
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Other Names | AlkB homolog 9 antibody, ALKBH9 antibody, Alpha-ketoglutarate-dependent dioxygenase FTO antibody, AW743446 antibody, Fat mass and obesity-associated protein antibody, FATSO, MOUSE, HOMOLOG OF antibody, Fto antibody, FTO_HUMAN antibody, GDFD antibody, KIAA1752 antibody, mKIAA1752 antibody, Protein fatso antibody |
Target/Specificity | The FTO gene is the most robust gene for common obesity characterized to date. FTO gene expression has been found to be significantly upregulated in the hypothalamus of rats after food deprivation and strongly negatively correlated with the expression of orexin peptide which is involved in the stimulation of food intake (Fredricksson R et al., 2008). Deletion analysis of FTO gene in mice showed that FTO is functionally involved in the control of both energy intake and energy expenditure (Fischer J et al., 2009). |
Format | Protein G Purified |
Storage | Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles. |
Precautions | Anti-FTO (Fat mass and obesity related protein) Antibody is for research use only and not for use in diagnostic or therapeutic procedures. |
Shipping | Blue Ice |

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Provided below are standard protocols that you may find useful for product applications.
Background
The FTO gene is the most robust gene for common obesity characterized to date. FTO gene expression has been found to be significantly upregulated in the hypothalamus of rats after food deprivation and strongly negatively correlated with the expression of orexin peptide which is involved in the stimulation of food intake (Fredricksson R et al., 2008). Deletion analysis of FTO gene in mice showed that FTO is functionally involved in the control of both energy intake and energy expenditure (Fischer J et al., 2009).

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